|
|
Human Endogenous retroviruses as potential early markers for Alzheimers disease |
| 3R01CA226570-04S1 |
|
NCI |
2022 |
|
|
Association of Tau and Neuronal Hypometabolism with Positron Emission Tomography in Alzheimers Disease |
| 1F30AG074524-01A1 |
|
NIA |
2022 |
|
|
Effects of metabolic phenotype on functional connectivity in aging and Alzheimer’s Disease |
| 1F30AG076278-01 |
|
NIA |
2022 |
|
|
Fluency from Flesh to Filament: Collation, Representation, and Analysis of Multi-Scale Neuroimaging data to Characterize and Diagnose Alzheimers Disease |
| 1F30AG077736-01 |
|
NIA |
2022 |
|
|
Multimodal, multiclass prediction of disease status in Alzheimer’s |
| 1F30AG079610-01 |
|
NIA |
2022 |
|
|
AMPK localization, expression, and activity in Alzheimers Disease |
| 1F30AG079633-01 |
|
NIA |
2022 |
|
|
Single-cell epigenomic roadmap of Alzheimers disease |
| 1F31AG076308-01 |
|
NIA |
2022 |
|
|
Pathologic Substrates of Neuropsychiatric Symptoms in Aphasic Dementia |
| 1F31AG076318-01 |
|
NIA |
2022 |
|
|
Development of Novel Synthetic Proteomimetics for Mediating Tauopathy in Alzheimers Disease |
| 1F31AG076334-01 |
|
NIA |
2022 |
|
|
The combinatorial effects of familial mutations, oxidative stress, and aging on BBB dysfunction in Alzheimers disease |
| 1F31AG077930-01 |
|
NIA |
2022 |